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	<title>Comments on: Lipo-Gate / Omega-3 Watergate</title>
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	<link>https://www.sunsyncnutrition.com/blog/?p=1594</link>
	<description>SunSync Nutrition</description>
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		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=1594&#038;cpage=1#comment-5453</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Wed, 14 Jun 2017 03:35:01 +0000</pubDate>
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		<description><![CDATA[Yellow Fat Disease is not a vitamin E deficiency any more than aspirin poisoning is an activated charcoal deficiency or mercury poisoning is a chelation deficiency.]]></description>
		<content:encoded><![CDATA[<p>Yellow Fat Disease is not a vitamin E deficiency any more than aspirin poisoning is an activated charcoal deficiency or mercury poisoning is a chelation deficiency.</p>
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		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=1594&#038;cpage=1#comment-5448</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Fri, 09 Jun 2017 22:00:29 +0000</pubDate>
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		<description><![CDATA[Swami Nitty-Gritty called &quot;liver spots&quot; (age spots) a &quot;mini-leprosy.&quot;

He called leprosy &quot;runaway liver spots.&quot;]]></description>
		<content:encoded><![CDATA[<p>Swami Nitty-Gritty called &#8220;liver spots&#8221; (age spots) a &#8220;mini-leprosy.&#8221;</p>
<p>He called leprosy &#8220;runaway liver spots.&#8221;</p>
]]></content:encoded>
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		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=1594&#038;cpage=1#comment-5447</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Fri, 09 Jun 2017 21:49:19 +0000</pubDate>
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		<description><![CDATA[A pale gold pigment accumulates in the bone marrow following certain diseases (esp. those involving fever).

The pigment is lipofuscin — Yellow Fat Disease.]]></description>
		<content:encoded><![CDATA[<p>A pale gold pigment accumulates in the bone marrow following certain diseases (esp. those involving fever).</p>
<p>The pigment is lipofuscin — Yellow Fat Disease.</p>
]]></content:encoded>
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	<item>
		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=1594&#038;cpage=1#comment-5446</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Fri, 09 Jun 2017 21:44:38 +0000</pubDate>
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		<description><![CDATA[Two ways to make cholesterol ...
 
1) saturated fat

2) simple sugars

PUFAs and HUFAs block cholesterol synthesis.

&lt;&gt;

Cancer doesn&#039;t burn sugar.

It transforms amino acids into sugar and then into fat, which it burns.

Cancer burns so HOT doctors can detect it by its heat signature.

&lt;&gt;

Cancer especially loves the amino acids glutamine and arginine.

Cancer walks big circles around uric acid.]]></description>
		<content:encoded><![CDATA[<p>Two ways to make cholesterol &#8230;</p>
<p>1) saturated fat</p>
<p>2) simple sugars</p>
<p>PUFAs and HUFAs block cholesterol synthesis.</p>
<p><></p>
<p>Cancer doesn&#8217;t burn sugar.</p>
<p>It transforms amino acids into sugar and then into fat, which it burns.</p>
<p>Cancer burns so HOT doctors can detect it by its heat signature.</p>
<p><></p>
<p>Cancer especially loves the amino acids glutamine and arginine.</p>
<p>Cancer walks big circles around uric acid.</p>
]]></content:encoded>
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	<item>
		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=1594&#038;cpage=1#comment-5445</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Fri, 09 Jun 2017 21:34:56 +0000</pubDate>
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		<description><![CDATA[D.A. Gray &amp; J. Woulfe (&quot;Lipofuscin and aging: a matter of toxic waste,&quot; Science of Aging Knowledge Environment, Feb. 2005) wrote ...
 
&quot;Lipofuscin is membrane-bound cellular waste that can be neither degraded nor ejected from the cell but can only be diluted through cell division and subsequent growth. The fate of postmitotic cells is to accumulate lipofuscin, which as an &#039;aging pigment&#039; has been considered a reliable biomarker for the age of cells such as neurons and, by extension, their hosts. In the aging human brain, deposits of lipofuscin are not uniformly distributed but are concentrated in specific regions of functional interest. The prevailing thought is that the major source of lipofuscin is incomplete lysosomal degradation of damaged mitochondria. Accumulating evidence suggests that lipofuscin is not benign but can impair the functioning of seemingly unrelated cellular systems, including the ubiquitin/proteasome pathway. A damaging feedback loop of lysosomal and proteasomal inhibition may occur as lipofuscin accumulates, leading to what has been appropriately named a &#039;garbage catastrophe.&#039; Reversing this catastrophe presents a formidable challenge.&quot;]]></description>
		<content:encoded><![CDATA[<p>D.A. Gray &#038; J. Woulfe (&#8220;Lipofuscin and aging: a matter of toxic waste,&#8221; Science of Aging Knowledge Environment, Feb. 2005) wrote &#8230;</p>
<p>&#8220;Lipofuscin is membrane-bound cellular waste that can be neither degraded nor ejected from the cell but can only be diluted through cell division and subsequent growth. The fate of postmitotic cells is to accumulate lipofuscin, which as an &#8216;aging pigment&#8217; has been considered a reliable biomarker for the age of cells such as neurons and, by extension, their hosts. In the aging human brain, deposits of lipofuscin are not uniformly distributed but are concentrated in specific regions of functional interest. The prevailing thought is that the major source of lipofuscin is incomplete lysosomal degradation of damaged mitochondria. Accumulating evidence suggests that lipofuscin is not benign but can impair the functioning of seemingly unrelated cellular systems, including the ubiquitin/proteasome pathway. A damaging feedback loop of lysosomal and proteasomal inhibition may occur as lipofuscin accumulates, leading to what has been appropriately named a &#8216;garbage catastrophe.&#8217; Reversing this catastrophe presents a formidable challenge.&#8221;</p>
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