They’re both chemotherapy for the gums and teeth. Allopathic medicine often backfires and makes things worse.

Our highest choice is to achieve a healthy oral ecology by eating foods (On Time is best) that build immunity (super-immunity is best) and keep the peace in the microbiome (the collective bacteriome, virome, and mycobiome).

They had almost no access to PUFAs, HUFAs, and foods high in estrogen, nitric oxide, serotonin, and iron.

Those who did have access, and there were many, paid the price, as forensic paleontologists have discovered.

Lasers are almost as aggressive as scraping. And red light therapy won’t work for calculus.

As long as they’re unprocessed, no problem.

Pine sap or turpentine taken directly from the tree are whole natural products.

Xylitol and high-fructose corn syrup are not only processed and GMO — they’re overly processed.

K.N. Sulochana, S. Ramakrishnan, S.B. Vasanthi, H.N. Madhavan, K. Arunagiri, & R. Punitham (“First report of congenital or infantile cataract in deranged proteoglycan metabolism with released xylose,” The British Journal of Ophthalmology, Apr. 1997) wrote …

“Of 220 children of both sexes below 12 years of age, with congenital or infantile cataract treated in Sankara Nethralaya, Madras, India, during a period of 2 years, 145 excreted fragments of GAG (heparan and chondroitin sulphates) in their urine. There was no such excretion among the control group of 50 children. The same was found accumulated in the blood and lenses of affected children. In addition, xylose was present in small amounts in the urine and blood and xylitol was present in the lens. There was a significant elevation in the activity of beta glucuronidase in lymphocytes and urine, when compared with normals. All the above findings suggest deranged proteoglycan metabolism. As the urine contained mostly GAG fragments and very little xylose, Benedict’s reagent was not reduced. This ruled out galactosaemia. CONCLUSION: An increase of beta glucuronidase activity might have caused extensive fragmentation of GAG with resultant accumulation in the blood and lens and excretion in urine. Small amounts of xylose may have come from xylose links between GAG and core protein of proteoglycans. Owing to their polyanionic nature, GAG fragments in the lens might abstract sodium, and with it water, thereby increasing the hydration of the lens. Excessive hydration and the osmotic effect of xylitol from xylose might cause cataract. While corneal clouding has been reported in inborn acid mucopolysaccharidosis, congenital or infantile cataract with deranged metabolism of proteoglycans (acid mucopolysaccharide-xylose-protein complex) is reported in children for the first time.”