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	<title>Comments on: Lipofuscin Degeneration</title>
	<atom:link href="https://www.sunsyncnutrition.com/blog/?feed=rss2&#038;p=2446" rel="self" type="application/rss+xml" />
	<link>https://www.sunsyncnutrition.com/blog/?p=2446</link>
	<description>SunSync Nutrition</description>
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		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=2446&#038;cpage=1#comment-8000</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Sat, 25 Jul 2020 23:45:53 +0000</pubDate>
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		<description><![CDATA[According to Adano Ley (Swami Nitty-Gritty) ...

&quot;Reflex therapy does not correct at the entrance point. There is more area to work at the exit point. The entrance point is smaller than the exit. This is analogous to a bullet wound.&quot;]]></description>
		<content:encoded><![CDATA[<p>According to Adano Ley (Swami Nitty-Gritty) &#8230;</p>
<p>&#8220;Reflex therapy does not correct at the entrance point. There is more area to work at the exit point. The entrance point is smaller than the exit. This is analogous to a bullet wound.&#8221;</p>
]]></content:encoded>
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		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=2446&#038;cpage=1#comment-7999</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Sat, 25 Jul 2020 23:43:23 +0000</pubDate>
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		<description><![CDATA[The sympathetic nervous system gets a bad rap.

It&#039;s always active at a basal metabolic level. This is called &quot;sympathetic tone.&quot;

You can&#039;t get out of bed or raise up from a chair without the cooperation of your sympathetic nervous system, let alone &quot;stand on your own two feet.&quot;]]></description>
		<content:encoded><![CDATA[<p>The sympathetic nervous system gets a bad rap.</p>
<p>It&#8217;s always active at a basal metabolic level. This is called &#8220;sympathetic tone.&#8221;</p>
<p>You can&#8217;t get out of bed or raise up from a chair without the cooperation of your sympathetic nervous system, let alone &#8220;stand on your own two feet.&#8221;</p>
]]></content:encoded>
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	<item>
		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=2446&#038;cpage=1#comment-7998</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Sat, 25 Jul 2020 23:36:12 +0000</pubDate>
		<guid isPermaLink="false">http://www.sunsyncnutrition.com/blog/?p=2446#comment-7998</guid>
		<description><![CDATA[Lipofuscin doesn&#039;t just sit passively in your cells, clogging them up.

It aggressively attacks both your cells and their milieu (microenvironment).]]></description>
		<content:encoded><![CDATA[<p>Lipofuscin doesn&#8217;t just sit passively in your cells, clogging them up.</p>
<p>It aggressively attacks both your cells and their milieu (microenvironment).</p>
]]></content:encoded>
	</item>
	<item>
		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=2446&#038;cpage=1#comment-7997</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Sat, 25 Jul 2020 23:31:59 +0000</pubDate>
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		<description><![CDATA[Here are a few things to expedite the elimination of volatile (and therefore rancid) fats, including the ones containing LIPOFUSCIN …

1) Sweet tropical fruit for breakfast (the sweeter the better)

2) Oranges at Spleen-Pancreas Time (9:00-11:00 a.m.) (Grapefruit is counterproductive)

3) Sweet berries at Urinary Bladder Time (3:00-5:00 p.m.)

4) Buttered and salted dextrinized potato for supper

5) Pineapple juice at Triple Heater Time (9:00-11:00 p.m.)]]></description>
		<content:encoded><![CDATA[<p>Here are a few things to expedite the elimination of volatile (and therefore rancid) fats, including the ones containing LIPOFUSCIN …</p>
<p>1) Sweet tropical fruit for breakfast (the sweeter the better)</p>
<p>2) Oranges at Spleen-Pancreas Time (9:00-11:00 a.m.) (Grapefruit is counterproductive)</p>
<p>3) Sweet berries at Urinary Bladder Time (3:00-5:00 p.m.)</p>
<p>4) Buttered and salted dextrinized potato for supper</p>
<p>5) Pineapple juice at Triple Heater Time (9:00-11:00 p.m.)</p>
]]></content:encoded>
	</item>
	<item>
		<title>By: sunsync Nutrition</title>
		<link>https://www.sunsyncnutrition.com/blog/?p=2446&#038;cpage=1#comment-7996</link>
		<dc:creator><![CDATA[sunsync Nutrition]]></dc:creator>
		<pubDate>Sat, 25 Jul 2020 23:28:12 +0000</pubDate>
		<guid isPermaLink="false">http://www.sunsyncnutrition.com/blog/?p=2446#comment-7996</guid>
		<description><![CDATA[G. Di Guardo (&quot;Lipofuscin, Lipofuscin-Like Pigments and Autofluorescence,&quot; European Journal of Histochemistry, Feb. 3, 2015) wrote ...

&quot;... lipofuscin and lipofuscin-like compounds should be regarded as aggregates of undigested cell materials resulting from phagocytosis and autophagy processes and accumulating as endocytoplasmic granules under both physiological and pathological conditions. During ageing, for instance, lipofuscin accumulation may be physiologically observed in the liver as well as in the central nervous system, at the level of which the occurrence of lipofuscin, lipofuscin-like lipopigments and ceroid may be also detected during oxidative stress, or as a response to given lysosomal storage diseases (Batten disease or neuronal ceroid lipofuscinosis) and to a number of physical and chemical noxae, such as radiation, cisplatin, lead (Pb) and mercury (methyl-Hg), under both natural and experimental disease conditions. Moreover, lipofuscin and lipofuscin-like substances, which alongside with their well known AF properties may be easily demonstrated in host cells by means of Schmorl and/or Sudan Black histochemical stains, have been reported to increase in human mesenchymal stem cells subjected to oxidative stress, while decreasing in murine embryonic stem cells undergoing differentiation, a finding that might be of relevance also in relation to the recently described epithelial CSC-specific AF. Beside what above, lipofuscin bodies may be observed in a number of additional pathologic conditions, both neoplastic and non-neoplastic, such as pancreatic tumours, non-choroidal melanomas, mammary gland carcinomas, pigmented eyelid cysts, retinal degenerative processes like age-related macular degeneration and brown bowel syndrome or intestinal lipofuscinosis.&quot;]]></description>
		<content:encoded><![CDATA[<p>G. Di Guardo (&#8220;Lipofuscin, Lipofuscin-Like Pigments and Autofluorescence,&#8221; European Journal of Histochemistry, Feb. 3, 2015) wrote &#8230;</p>
<p>&#8220;&#8230; lipofuscin and lipofuscin-like compounds should be regarded as aggregates of undigested cell materials resulting from phagocytosis and autophagy processes and accumulating as endocytoplasmic granules under both physiological and pathological conditions. During ageing, for instance, lipofuscin accumulation may be physiologically observed in the liver as well as in the central nervous system, at the level of which the occurrence of lipofuscin, lipofuscin-like lipopigments and ceroid may be also detected during oxidative stress, or as a response to given lysosomal storage diseases (Batten disease or neuronal ceroid lipofuscinosis) and to a number of physical and chemical noxae, such as radiation, cisplatin, lead (Pb) and mercury (methyl-Hg), under both natural and experimental disease conditions. Moreover, lipofuscin and lipofuscin-like substances, which alongside with their well known AF properties may be easily demonstrated in host cells by means of Schmorl and/or Sudan Black histochemical stains, have been reported to increase in human mesenchymal stem cells subjected to oxidative stress, while decreasing in murine embryonic stem cells undergoing differentiation, a finding that might be of relevance also in relation to the recently described epithelial CSC-specific AF. Beside what above, lipofuscin bodies may be observed in a number of additional pathologic conditions, both neoplastic and non-neoplastic, such as pancreatic tumours, non-choroidal melanomas, mammary gland carcinomas, pigmented eyelid cysts, retinal degenerative processes like age-related macular degeneration and brown bowel syndrome or intestinal lipofuscinosis.&#8221;</p>
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