Traumatic shock promotes systemic catabolism (hyper-alkalinity), a situation capable of decreasing the growth of cancer (a hyper-acidic condition) but increasing the metastasis of cancer (a hyper-alkaline condition).

Nathan Seppa (“Pancreatic enzymes may play role in shock,” Science News, Feb. 19, 2000) wrote …

“By blocking the effects of pancreatic enzymes, researchers at the University of California, San Diego in La Jolla prevented shock in rats prone to get it — the first solid evidence that points to enzymes as catalysts for this condition.”

Emanuel Revici, M.D. (Research In Physiology As Basis Of Guided Chemotherapy: With Special Application To Cancer, 1961) wrote …

“Since the various changes in the state of shock affect the blood and two organs, the stomach and duodenum, they can be considered to involve the organic and systemic levels.”